Salk Institute: SARS-CoV-2 Spike Proteins Cause Damage To Blood Vessels But the Same Spike Proteins Coded By the Vaccines Are Safe!

We already knew that the SARS-CoV-2 spike proteins were implicated in serious damage to blood vessels in severe cases of Covid disease in the immune-compromised. Here (Sept 2020) for instance:

Critically ill patients diagnosed with COVID-19 may develop a pro-thrombotic state that places them at a dramatically increased lethal risk.

We demonstrated that COVID-19 patients present with increased mean platelet volume (MPV) and platelet hyperactivity, which correlated with a decrease in overall platelet count. Detectable SARS-CoV-2 RNA in the blood stream was associated with platelet hyperactivity in critically ill patients. Platelets expressed ACE2, a host cell receptor for SARS-CoV-2, and TMPRSS2, a serine protease for Spike protein priming. SARS-CoV-2 and its Spike protein directly enhanced platelet activation such as platelet aggregation, PAC-1 binding, CD62P expression, α granule secretion, dense granule release, platelet spreading, and clot retraction in vitro, and thereby Spike protein enhanced thrombosis formation in wild-type mice transfused with hACE2 transgenic platelets, but this was not observed in animals transfused with wild-type platelets in vivo.

Our findings uncovered a novel function of SARS-CoV-2 on platelet activation via binding of Spike to ACE2. SARS-CoV-2-induced platelet activation may participate in thrombus formation and inflammatory responses in COVID-19 patients.

Or here (Jan 2021):

It was found that the treatment of cultured primary human pulmonary artery smooth muscle cells (SMCs) or human pulmonary artery endothelial cells with the recombinant SARS-CoV-2 spike protein S1 subunit is sufficient to promote cell signaling without the rest of the viral components [21]. Furthermore, our analysis of the postmortem lung tissues of patients who died of COVID-19 has determined that these patients exhibited pulmonary vascular wall thickening, a hallmark of pulmonary arterial hypertension (PAH) [21]. Based on these results, we proposed that the SARS-CoV-2 spike protein (without the rest of the viral components) triggers cell signaling events that may promote pulmonary vascular remodeling and PAH as well as possibly other cardiovascular complications [21,22].

The Salk Institute have recently published another study which comes to much the same conclusions. They seem to think they might be the first to discover the mechanism whereby the spike causes endothelial damage:

The paper, published on April 30, 2021, in Circulation Research, also shows conclusively that COVID-19 is a vascular disease, demonstrating exactly how the SARS-CoV-2 virus damages and attacks the vascular system on a cellular level. The findings help explain COVID-19’s wide variety of seemingly unconnected complications, and could open the door for new research into more effective therapies.

“A lot of people think of it as a respiratory disease, but it’s really a vascular disease,” says Assistant Research Professor Uri Manor, who is co-senior author of the study. “That could explain why some people have strokes, and why some people have issues in other parts of the body. The commonality between them is that they all have vascular underpinnings.”

While the findings themselves aren’t entirely a surprise, the paper provides clear confirmation and a detailed explanation of the mechanism through which the protein damages vascular cells for the first time. There’s been a growing consensus that SARS-CoV-2 affects the vascular system, but exactly how it did so was not understood. Similarly, scientists studying other coronaviruses have long suspected that the spike protein contributed to damaging vascular endothelial cells, but this is the first time the process has been documented.

Previous studies have shown a similar effect when cells were exposed to the SARS-CoV-2 virus, but this is the first study to show that the damage occurs when cells are exposed to the spike protein on its own.

Just looking at the other two studies cited above (which are by no means exhaustive) I think Salk’s claim to originality is somewhat dubious. But what is even more dubious is their claim (minus any proper scientific evidence) that the spike protein of the virus itself can cause serious cardiovascular symptoms independent of the virus in those infected with SARS-CoV-2 but that, for some mysterious reason, the very same spike proteins generated by the body’s cells after vaccination are ‘safe’ and do not cause such symptoms! Hence they say:

LA JOLLA—Scientists have known for a while that SARS-CoV-2’s distinctive “spike” proteins help the virus infect its host by latching on to healthy cells. Now, a major new study shows that the virus spike proteins (which behave very differently than those safely encoded by vaccines) also play a key role in the disease itself.

Excuse me? The scientific justification for this claim appears to come from this brief and somewhat confusing statement from the authors themselves:

. . . . . . our data reveals that S protein alone can damage endothelium, manifested by impaired mitochondrial function and eNOS activity but increased glycolysis. It appears that S protein in ECs increases redox stress which may lead to AMPK deactivation, MDM2 upregulation, and ultimately ACE2 destabilization.4 Although these findings need to be confirmed with the SARS-CoV-2 virus in the future study, it seems paradoxical that ACE2 reduction by S protein would decrease the virus infectivity, thereby protecting endothelium. However, a dysregulated renin-angiotensin system due to ACE2 reduction may exacerbate endothelial dysfunction, leading to endotheliitis. Collectively, our results suggest that the S protein-exerted EC damage overrides the decreased virus infectivity. This conclusion suggests that vaccination-generated antibody and/or exogenous antibody against S protein not only protects the host from SARS-CoV-2 infectivity but also inhibits S protein-imposed endothelial injury.

Does that mealy-mouthed scientific explanation make sense? It doesn’t to me. I’m not an expert admittedly but I can sense there is something amiss here. Especially considering the fact that other scientists have also questioned the wisdom of injecting young, healthy people with a ‘vaccine’ which induces their body cells to manufacture the very spike protein which, in severe cases of Covid – which young, healthy people are at very minimal risk from – causes life-threatening cardiovascular disease. Hence, the authors of the PubMed study cited above say:

Vaccines that introduce the spike protein into our body to elicit virus-neutralizing antibodies are currently being developed. In this article, we note that human host cells sensitively respond to the spike protein to elicit cell signaling. Thus, it is important to be aware that the spike protein produced by the new COVID-19 vaccines may also affect the host cells. We should monitor the long-term consequences of these vaccines carefully, especially when they are administered to otherwise healthy individuals.

But, you know, vaccine passport, idiot selfish refuseniks and all that. They’ve simply got to jab every single person on the planet, including your kids, because ‘nobody’s safe until we’re all safe’, right? With the huge and growing number of documented cardiovascular adverse reactions to the ‘vaccines’ (heart attack, stroke, eye damage, haemorrhaging, blood clots, ‘extremely rare’ cerebral haemorrhaging, heavy periods in women) I think it is a near certainty that the spike proteins raised by the ‘vaccines’ do not behave differently at all from the spike protein of the SARS-CoV-2 virus itself and the only difference is that the ‘vaccine’ spike proteins are causing severe cardiovascular disease in people who would not be at significant risk of severe cardiovascular symptoms from SARS-CoV-2 infection, because their innate immune system would prevent the virus from ever progressing to the point where it was able to enter the blood stream and effectively poison the blood with toxic spikes.


It gets murkier. Robin Monotti on Telegram made a Youtube video about this Salk study which they promply deleted, calling it ‘medical misinformation’. What a surprise! However, what did surprise me is that Salk did not originally claim the ‘vaccine’ spike proteins were safe in their initial press release and they actually changed the wording in the article after Robin had released his video. No conspiracy there then.

Note that since I released this video Salk added the words “safely encoded” in their article in their also newly added description of the vaccine spike protein. VAERS numbers indicate this theoretical description does not correspond to the full and complete reality of the situation. Original Salk text was this: “Scientists have known for a while that SARS-CoV-2’s distinctive “spike” proteins help the virus infect its host by latching on to healthy cells. Now, a major new study shows that they also play a key role in the disease itself”

The video is available here on Brandnewtube:

And on Bitchute:

They really, really, really want you to get jabbed with these ‘vaccines’.

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